A patient under treatment for “cirrhosis and ascites”. Can you tell what is being demonstrated in colour M mode through hepatic vein.

For opinion. 30 yr old female,G2P1, now 32 weeks gestation. Post TOF ICR with transannular patch in childhood, has severe PR, TR.Class II DOE now. JVP shown.With lasix she feels better. How to manage?Advised hospitalisation.Will you ask obstetric dept to Induce delivery or wait?

58 yr old male, secundum ASD 34 mm adv surgery previously, lost to follow up and presented with HF after onset of AF. ASD shunting left to right shunt, Severe TR, MR as shown in A4C. Cath showed LVEDP18, QP/QS 2.2:1, RVSP 62. LAD total occlusion, OM&RCA tight lesions. Your advise

Name of Actinobacillus changed to Aggregatibacter!!Why they change the name of bacteria so frequently, to cause hardships to we poor guys?!😄🤭To change our names it takes hundreds of formalities here.everything from driving licence,Aadhar card, passport,voter id card suffers

If you don’t see much MR in standard views, never forget to assess MR in short axis view, especially in RHD. Take short axis of MV, put colour and sweep the probe gently up and down. Commissural MR and valvular MR can be clearly seen. This pt had minimal MR in other views.

Just see a so prominent delta wave. See V2, is that a separate delta wave leading to that funny QRS?!! Any significance for this appearance? ⁦Syamkumar⁩ ⁦Dr Ulhas Pandurangi⁩

For fellows. Pacemaker done in another center, patient came to us with recurrence of syncope. What are the findings? Likely mistakes happened during initial procedure?

Is it clot or pectinate muscle in RA appendage? In LA appendage, velocity in LAA may be a useful clue, if normal more likely pectinate muscle. Anything like that in RAA other than ICE? We can’t afford ICE